Akvatsitramon

Description overdue on 10/14/2013

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Combined preparation analgesic . It has anti-inflammatory, antipyretic, analgesic effects. The drug inhibits blood clots, reduces the rate of platelet aggregation, increases microcirculation in the area of ​​inflammatory focus. Active ingredients: acetylsalicylic acid, caffeine, paracetamol. Caffeine   has a stimulating effect on the vasomotor and respiratory centers, increases the excitability of spinal cord reflex level, expands the lumen of the arteries in the kidney, skeletal muscle, heart, brain. Caffeine improves performance (physical and umtsvennaya), reduces the severity of fatigue, excessive sleepiness, reduces platelet aggregation rate of cells.

The preparation Akvatsitramon caffeine contained in the minimum dose that increases blood flow and vascular tonus normalized, but does not stimulate the central nervous system. Paracetamol   It has anti-inflammatory effects; additionally has antipyretic and analgesic effects. This effect of paracetamol due to its ability to inhibit lipid synthesizing prostaglandins in the tissues located peripherally. Antipyretic effect of paracetamol provided its impact on heat regulating center   in the hypothalamus. Akvatsitramon it produced in the form of capsules, granules, a tablet dosage form. Otpuskaetsya without prescription form.

Indications:

Akvatsitramon prescribed for arthralgia , Toothache, algodismenoree, myalgia, neuralgia, migraine, headache. Drug Usage Akvatsitramon recommends combination drug for relief pain   (mild, moderate). The drug is prescribed for flu and ARI for relief febrile syndrome .

Contraindications:

Akvatsitramon not prescribed for Hemophilia , In lesions of the digestive tract in the acute stage (erosive and ulcerative in nature), while vitamin deficiency of vitamin K, portal hypertension, gipoprotrombinemii, with "aspirin" asthma, hemorrhagic diathesis, breastfeeding, severe coronary artery disease, with severe portal hypertension , Violation of sleep, of increased excitability, renal failure, glaucoma. Babies Akvatsitramon contraindicated because of the risk of Reye's syndrome (on the background of flowing viral disease). In the pathology of the liver (hepatitis, cirrhosis), gout drug is prescribed with caution in the subacute phase of the inflammatory process. Aspirin, which is part of Akvatsitramona, has teratogenic effects on the fetus . In depending on the term pregnancy are formed different the vices of development. First trimester: the splitting of the upper sky. Third trimester: the closure of the ductus arteriosus in the fetus (hypertension in the pulmonary circulation, pulmonary vascular hyperplasia), inhibition of labor (due to inhibition of lipid synthesizing prostaglandins). The active component allocated with thoracal milk, increasing the risk of bleedings in children because of the of a functional violations in the thrombocyte cells.

Side effect:

Akvatsitramon can cause allergic responses, bronchospasm, vomiting, nausea, increased blood pressure. Chronic administration of the drug observed dizziness,   hypocoagulation , Decrease in the rate aggregation thrombocyte cells, headache, noise in the ears, violations of of visual perception, hemorrhagic syndrome , Nosebleeds, defeat of the digestive tract (ulcerative erosive character), tachycardia, purpura, toxic necrolysis , Exudative erythema malignant course, Reye's syndrome (vomiting, disorders of the psyche, functional changes in the liver, metabolic acidosis, hyperpyrexia).

Overdose:

Lungs intoxication:   ringing at the Shah, stomachalgia , Nausea, dizziness, vomiting (due to the toxic effects of acetylsalicylic acid). In severe poisoning   notes anuria , Somnolence, bronchoconstriction, convulsions, bleeding, difficulty in breathing, collapse, confusion of consciousness. Initially, the administration is central hyperventilation respiratory alkalosis   (cyanosis of the extremities, asthma, shortness of breath, sweating). Further, the severity of intoxication increases, which leads to the progression of respiratory paralysis, to the disunity of oxidative phosphorylation and ultimately formed carbon dioxide acidosis . If poisoning Akvatsitramonom recommended administration lactate, sodium citrate or sodium (depending on the state of metabolism). It requires the constant control over the main indicators of electrolyte balance   and acid-base status. Withdrawal of one of the components medikament- acetylsalicylic acid increases with rising reserve alkalinity by alkalinization of urine.

Mode of application:

Akvatsitramon taken orally. For the relief of pain appoint 1-2 tablets. The average daily dose - 3-4 tablets. The drug is recommended to be taken with a meal or after. The maximum number of tablets per day - 8 pieces. Duration of treatment - no more than 7-10 days. The drug should not be taken for more than 3 days for relief of febrile syndrome and reduce the temperature; more than 5 days - for pain relief. The treatment regimen and dosing of the drug should be agreed with your physician.

Special instructions:

Illegal assignment Akvatsitramona children, because of the risk of Reye's syndrome   (reaction to acetylsalicylic acid in viral diseases): the formation of an acute encephalopathy, enlarged liver, uncontrollable vomiting. Long-term treatment requires mandatory Akvatsitramonom control enzymes ALT, AST. It is necessary to warn the anesthesiologist and the surgeon about the medications before surgery. Patients with wheeze reactions and hypersensitivity to salicylates drug can be administered only in a hospital. Low dose aspirin slows down the excretion of uric acid (important to consider when treating patients with gout). Because of the risk of gastrointestinal bleeding unacceptable use ethanol Akvatsitramonom during treatment.

Drug Interactions:

The rate of absorption ergotamine   increases during treatment Akvatsitramonom.
The drug enhances the effect of steroid hormone , Heparin, hypoglycemic agents, anticoagulants, reserpine . When concurrent therapy Methotrexate   and other NSAIDs there is a growing severity of their side effects. The medicament reduces efficiency of furosemide , Spironolactone, antihypertensive and protivopodagricakih drugs that increase excretion Uric acid excreted. Salicylamide, rifampicin , Barbiturates, stimulants microsomal oxidation, antiepileptic drugs increase the formation of toxic metabolites of paracetamol, which have a detrimental effect on the work of the hepatic system. Absorption of paracetamol in the treatment of accelerated metoclopramide . The duration of excretion chloramphenicol increases 5 times when receiving Akvatsitramona. Action derivatives bishydroxycoumarin ( anticoagulants ) Amplifies at repeated reception of paracetamol. Hepatotoxic the impact of   the drug increases while taking ethanol and paracetamol.